Authors:

PATTERSON, K. & LAMBON RALPH, M.A.

Reference:

Year of publication:

1999

CBU number:

3868

Abstract:

Over the past few decades, refined cognitive architectures with highly specific components have been proposed to explain apparently selective disorders of reading, resulting from brain disease or injury, in previously literate adults. Recent analysis of the more general linguistic and cognitive abilities supported by neural systems damages in the various forms of alexia favours a rather different view of reading and the kinds of models sufficient to account for its acquisition, skilled performance and disruption.The hypothesis proposed in this paper is that for each of the major patterns of acquired alexia, there is a consistent accompanying impairment in another cognitive domain, and that the nature of both the reading and the associated nonreading disorder is predictable from the location of the brain lesion.